Seems to me it is the subsequent bacterial/fungal infection that is the true cause of death. And often acquired in-hospital or care facility. Any respiratory virus increases susceptibility. Some more than others. Stay out of the hospital if at all possible.
Why do you say unknown quantity of antigen? Surely the adeno viral vector is 1 spike protein per viral particle, and the mRNA is possibly more than 1 antigen per LNP, depending on how stable the LNP is for multiple translations, but nevertheless would be a fixed number of spike proteins per LNP??
Thanks for that. A Joseph Mengele thought here: maybe this period of global EUA was a necessary trial of the inefficacy of gene-based vaccines for coronas? How else would we ever have found out?
A possible reason that the immune system doesn't invest too much firepower in long-term immunity to very mild respiratory viruses is that memory b and t cells are an inefficient use of resources, which are needed to bekept in reserve for more serious pathogens. We've always known that immunity to colds is "sloppy".
He talks about the 'mucosal' immune system controlled by the gut, and the immune suppression that occurs to ensure that we don't treat everything we eat as pathogenic. Being a disease of the airways, OC43 will encounter the mucosal system, and will likely be treated by a system being deliberately suppressed. Antibodies are produced to prevent severe disease, but a desensitisation to the pathogen seems to take place. He believes that this is why the boosters are now failing in ever decreasing timescales: the immune response is being sensitised. Lots of antibodies, but lots of infection.
Oh no, this isn't going to drag on for 130 years plus is it!? Interesting info though, thanks!
https://nakedemperor.substack.com/
Thhis was the big debate between Dawkins and Stephen Jay Gould.
Imagine if someone said this in early 2020 and people understood it, rather than fearmongering for 2 years? What a travesty
I knew Fauci is old, but he was doing gain of function in the late 1800s? That can't be true, can it?
did you see the study in the Nursing home in Washington where they thought it was Sars but it was a common CV from some years back?
Seems to me it is the subsequent bacterial/fungal infection that is the true cause of death. And often acquired in-hospital or care facility. Any respiratory virus increases susceptibility. Some more than others. Stay out of the hospital if at all possible.
Everyone knows that the deadlier thing out there is the DNA... RNA is just fun. So all these "virus" are just to keep us entertained and distracted.
The combination of spermatozoon DNA with an egg DNA will kill the animal no matter what.
No amount of drugs, surgeries, amputations, DNA techniques, mRNA mambo-jambo jabs can avoid the power that the DNA possess to kill the Host.
Why do you say unknown quantity of antigen? Surely the adeno viral vector is 1 spike protein per viral particle, and the mRNA is possibly more than 1 antigen per LNP, depending on how stable the LNP is for multiple translations, but nevertheless would be a fixed number of spike proteins per LNP??
Thanks for that. A Joseph Mengele thought here: maybe this period of global EUA was a necessary trial of the inefficacy of gene-based vaccines for coronas? How else would we ever have found out?
A possible reason that the immune system doesn't invest too much firepower in long-term immunity to very mild respiratory viruses is that memory b and t cells are an inefficient use of resources, which are needed to bekept in reserve for more serious pathogens. We've always known that immunity to colds is "sloppy".
"Quite why the immune system prefers to take this approach to coronavirus (and some other upper respiratory tract infections) remains unanswered."
Perhaps we do know the answer to this. Take a look at this talk by Prof. Robert Clancy.
https://www.youtube.com/watch?v=FPPnyzvO7J4
He talks about the 'mucosal' immune system controlled by the gut, and the immune suppression that occurs to ensure that we don't treat everything we eat as pathogenic. Being a disease of the airways, OC43 will encounter the mucosal system, and will likely be treated by a system being deliberately suppressed. Antibodies are produced to prevent severe disease, but a desensitisation to the pathogen seems to take place. He believes that this is why the boosters are now failing in ever decreasing timescales: the immune response is being sensitised. Lots of antibodies, but lots of infection.